Symptoms of early symptoms appear wilting, cut open the diseased stems, tuber vascular bundle brown, humidity is high, the disease often produces white to pink hyphae.
The pathogen Fusarium oxysporum Schlecht. (Schlecht.) is called Fusarium spp. The pedestal is gray-brown; large conidiospores are formed in the sub-cones or conidiospores, sickle-shaped, curved, podocytes at base, 3 septa, size 19-452.5-5 (μm), 5-septate The size is 30-603.5-5 (μm). Small conidia 1 to 2 cells, oval or kidney-shaped, size 5-262-4.5 (μm), scattered in the mycelium, generally not mixed with large conidia. The chlamydospores are spherical, smooth or pleated, mostly single-celled, terminal or metastable, 5–15 μm in size. The bacteria can also infect tomatoes, bulbs, incense, melon, straw and so on.
Transmission Pathways and Pathogenic Conditions The pathogenic bacteria are mycelia or chlamydospores that overwinter with the diseased body in the soil or on the diseased diseased potato. The conidia produced in the diseased part of the year are transmitted through rain or irrigation water and infiltrated from the wound. The humidity in the field is large, the soil temperature is higher than 28°C, or it is easy to develop on low-lying land.
Prevention methods (1) Four-year rotation with grass crops or green manure. (2) The selection of sweet potatoes to retain seed, the application of decomposed organic fertilizer, and the strengthening of water and fertilizer management can reduce the incidence. (3) If necessary, water the 12.5% ​​synergistic carbendazim solvent 300 times.
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